{"id":12121,"date":"2021-01-11T11:05:53","date_gmt":"2021-01-11T13:05:53","guid":{"rendered":"https:\/\/mindthegraph.com\/blog\/?p=12121"},"modified":"2023-01-05T14:52:03","modified_gmt":"2023-01-05T17:52:03","slug":"cause-of-alzheimers-disease","status":"publish","type":"post","link":"https:\/\/mindthegraph.com\/blog\/hu\/cause-of-alzheimers-disease\/","title":{"rendered":"\u00daj elm\u00e9let pr\u00f3b\u00e1lja megmagyar\u00e1zni az Alzheimer-k\u00f3r ok\u00e1t"},"content":{"rendered":"

Napjainkban a demenciaesetek t\u00f6bb mint fel\u00e9t az Alzheimer-k\u00f3rhoz sorolj\u00e1k - ez egy kr\u00f3nikus neurodegenerat\u00edv betegs\u00e9g, amely fokozatosan befoly\u00e1solja az ember megismer\u0151 k\u00e9pess\u00e9geit \u00e9s mem\u00f3ri\u00e1j\u00e1t. <\/p>\n\n\n\n

Eg\u00e9szen napjainkig \u00fagy gondolt\u00e1k, hogy genetikai \u00e9s \u00f6reged\u00e9si t\u00e9nyez\u0151k okozz\u00e1k, a betegs\u00e9g gonosztev\u0151inek \u00e9s f\u0151 b\u0171n\u00f6seinek az \u00f6regkori plakkokat \u00e9s a neurofibrill\u00e1ris csom\u00f3sod\u00e1st tartj\u00e1k, ami neuroinflamm\u00e1ci\u00f3hoz \u00e9s sorban az idegsejtek elhal\u00e1s\u00e1hoz vezet. <\/p>\n\n\n\n

Az Alzheimer-k\u00f3r okair\u00f3l sz\u00f3l\u00f3 \u00faj elm\u00e9let azonban f\u00e9nyt der\u00edt az orvosi kutat\u00e1sok eg\u00e9re. <\/p>\n\n\n\n

Az elm\u00e9let szerint nem csak a genetika \u00e9s az \u00f6reged\u00e9s a felel\u0151s az amiloid csom\u00f3k - az Alzheimer-k\u00f3rt okoz\u00f3, az idegsejtek k\u00f6z\u00f6tt felhalmoz\u00f3d\u00f3 feh\u00e9rje - kialakul\u00e1s\u00e1\u00e9rt, hanem az is lehet, hogy a mikrobafert\u0151z\u00e9sre adott v\u00e1laszreakci\u00f3 is kiv\u00e1ltja.  <\/p>\n\n\n\n

N\u00e9zz\u00fcnk meg k\u00f6zelebbr\u0151l egy neurol\u00f3giai sk\u00e1l\u00e1t, hogy meg\u00e9rts\u00fck a teljes k\u00e9pet arr\u00f3l, mi t\u00f6rt\u00e9nik az Alzheimer-k\u00f3ros agyban. K\u00e9t f\u0151 jelens\u00e9g zajlik.<\/p>\n\n\n\n

A tau nev\u0171 feh\u00e9rje felel\u0151s a stabilit\u00e1s fenntart\u00e1s\u00e1\u00e9rt az\u00e1ltal, hogy a neuronok belsej\u00e9ben l\u00e9v\u0151 mikrotubulusok sz\u00e1m\u00e1ra t\u00e1maszt ny\u00fajt. A betegs\u00e9gben szenved\u0151 agyban a tau biok\u00e9miailag m\u00f3dosul a hiperfoszforil\u00e1t reakci\u00f3nak nevezett reakci\u00f3val. <\/p>\n\n\n\n

Ennek a v\u00e1ltoz\u00e1snak az eredm\u00e9nye, hogy az idegsejtek belsej\u00e9ben oldhatatlan feh\u00e9rje darabok keletkeznek, amelyek lebontj\u00e1k az eg\u00e9sz rendszert \u00e9s elpuszt\u00edtj\u00e1k az idegsejteket. <\/p>\n\n\n\n

A tau feh\u00e9rj\u00e9k ezen oldhatatlan darabjai egym\u00e1shoz aggreg\u00e1l\u00f3dnak, \u00e9s a tau feh\u00e9rj\u00e9ket alkotj\u00e1k. neurofibrill\u00e1ris csom\u00f3k.<\/strong><\/p>\n\n\n

\n
\"\"<\/a><\/figure><\/div>\n\n\n

A szenilis plakkok<\/strong> a k\u00f6vetkez\u0151kb\u0151l \u00e1llnak amiloid-\u03b2 peptidek<\/strong>. A neuronok membr\u00e1njaiban jelen l\u00e9v\u0151 feh\u00e9rje (pre-amyloid) hasad\u00e1si folyamaton megy kereszt\u00fcl, amelynek eredm\u00e9nyek\u00e9ppen az amiloid-\u03b2 szabad darabjai a neuronokon k\u00edv\u00fclre ker\u00fclnek*. <\/p>\n\n\n\n

Ezeket a darabokat elvileg speci\u00e1lis sejteknek \u00e9s enzimeknek kellene megtiszt\u00edtaniuk a k\u00fcls\u0151 neuronk\u00f6zegb\u0151l fagocit\u00f3zis \u00fatj\u00e1n, de genetikai vagy \u00f6reged\u00e9si t\u00e9nyez\u0151k miatt ez a folyamat nem t\u00f6rt\u00e9nik el\u00e9g hat\u00e9konyan, ami az oldhatatlan amiloid-\u03b2 feh\u00e9rj\u00e9k szabad darabjaihoz vezet, amelyek plakkokkokk\u00e1 kezdenek aggreg\u00e1l\u00f3dni.<\/p>\n\n\n

\n
\"\"<\/a><\/figure><\/div>\n\n\n

Az amiloid plakkok azok, amelyeket a legt\u00f6bbet tanulm\u00e1nyoztak \u00e9s vitattak az Alzheimer-k\u00f3r okak\u00e9nt. A tud\u00f3sok azonban azt is meger\u0151s\u00edtik, hogy a neurofibrill\u00e1ris csom\u00f3k k\u00f6zvetlen\u00fcl kapcsol\u00f3dnak a betegs\u00e9g el\u0151rehalad\u00e1s\u00e1hoz.<\/p>\n\n\n

\n
\"\"<\/a><\/figure><\/div>\n\n\n

A tud\u00f3sok eg\u00e9szs\u00e9ges pos-mortem agyakban is tal\u00e1ltak amiloid csom\u00f3t. Ami azt jelenti, hogy egy eg\u00e9szs\u00e9ges \u00f6reg agyban is kev\u00e9sb\u00e9 lesz hat\u00e9kony a tisztul\u00e1si folyamat, de nem annyira, mint egy Alzheimer-k\u00f3ros agyban.<\/p>\n\n\n\n

Ez\u00e9rt az Alzheimer-k\u00f3rra a nagysz\u00e1m\u00fa szenilis plakkok \u00e9s neurofibrill\u00e1ris csom\u00f3k jellemz\u0151ek; ezek rendk\u00edv\u00fcl m\u00e9rgez\u0151v\u00e9 v\u00e1lnak az agy sz\u00e1m\u00e1ra, \u00e9s megv\u00e1ltoztatj\u00e1k a szinapszisok hat\u00e9konys\u00e1g\u00e1t \u00e9s min\u0151s\u00e9g\u00e9t. <\/p>\n\n\n\n

Az \u00e9rintettek olyan t\u00fcneteket mutatnak, mint a mem\u00f3riaveszt\u00e9s, az id\u0151vel \u00e9s a helyzettel kapcsolatos zavarts\u00e1g, depresszi\u00f3, szorong\u00e1s, nyelvi \u00e9s kommunik\u00e1ci\u00f3s probl\u00e9m\u00e1k, hangulati instabilit\u00e1s \u00e9s m\u00e1sok.<\/p>\n\n\n\n

Az \u00faj javasolt elm\u00e9letben<\/strong>, az amiloid plakkok az agy mikrobafert\u0151z\u00e9ssel szembeni v\u00e9delmi mechanizmus\u00e1t jelentik. <\/p>\n\n\n\n

Az amiloid-\u03b2 g\u00e9n a gerincesek nagy r\u00e9sz\u00e9n\u00e9l jelen van, \u00e9s t\u00f6bb gener\u00e1ci\u00f3n kereszt\u00fcl megmaradt a k\u00fcl\u00f6nb\u00f6z\u0151 fajokban, ami azt jelenti, hogy hasznos \u00e9s fontos szerepe kell, hogy legyen a szervezet\u00fcnk sz\u00e1m\u00e1ra, \u00e9s nem csak mint valami k\u00e1ros dolog - ha nem lenne valami \u00e9rv\u00e9nyes vagy hasznos, akkor a g\u00e9n m\u00e1r r\u00e9g elt\u0171nt volna az evol\u00faci\u00f3s \u00faton.<\/p>\n\n\n\n

A tud\u00f3sok Alzheimer-k\u00f3ros emberek t\u00f6bb ezer post mortem agy\u00e1t vizsg\u00e1lt\u00e1k meg. <\/p>\n\n\n\n

B\u00e1r a rossz k\u00f6r\u00fclm\u00e9nyek miatt neh\u00e9zs\u00e9gekbe \u00fctk\u00f6ztek az ilyen t\u00edpus\u00fa agyak elemz\u00e9se sor\u00e1n, k\u00e9pesek voltak tanulm\u00e1nyozni \u00e9s inform\u00e1ci\u00f3t gy\u0171jteni, hogy k\u00e9s\u0151bb n\u00e9h\u00e1ny lehets\u00e9ges mikroba kiv\u00e1lt\u00f3 okot javasolhassanak. <\/p>\n\n\n\n

Ezek a kiv\u00e1lt\u00f3 t\u00e9nyez\u0151k a hum\u00e1n herpeszv\u00edrusok egyes t\u00edpusai, p\u00e9ld\u00e1ul a herpeszv\u00edrus 1, 6A \u00e9s 7, valamint h\u00e1rom bakt\u00e9rium elleni v\u00e9dekez\u00e9si mechanizmusok, Chlamydia pneumonia<\/em>, Borrelia burgdorferi<\/em>, Porphyromonas gingivalis<\/em>.<\/p>\n\n\n\n

Az elm\u00e9let szerint a b\u00e9ta-amiloid kezdetben v\u00e9dekez\u0151 funkci\u00f3t t\u00f6lt be, csapd\u00e1ba ejtve az agy fel\u00e9 mozg\u00f3 mikrob\u00e1kat, meg\u00e1ll\u00edtva egy esetleges fert\u0151z\u00e9st vagy betegs\u00e9get. <\/p>\n\n\n\n

Mint eml\u00edtett\u00fck, az \u00f6reged\u00e9s t\u00e9nyez\u0151i miatt az agy elvesz\u00edti k\u00e9pess\u00e9g\u00e9t az amiloidok hat\u00e9kony tiszt\u00edt\u00e1s\u00e1ra. \u00cdgy az amiloid funkci\u00f3j\u00e1r\u00f3l alkotott elk\u00e9pzel\u00e9s mindv\u00e9gig t\u00e9ves lehet; val\u00f3sz\u00edn\u0171leg egyfajta antimikrobi\u00e1lis v\u00e9delemben vesz r\u00e9szt az agyi betolakod\u00f3kkal szemben, mivel antimikrobi\u00e1lis peptidnek min\u0151s\u00fcl.<\/p>\n\n\n\n

A tud\u00f3sok pozit\u00edv eredm\u00e9nyeket \u00e9rtek el, amikor megpr\u00f3b\u00e1lt\u00e1k bizony\u00edtani, hogy a \u03b2-amiloid k\u00e9pes elpuszt\u00edtani a mikroorganizmusokat n\u00e9h\u00e1ny gyakori betegs\u00e9gben. <\/p>\n\n\n\n

Ezenk\u00edv\u00fcl egy m\u00e1sik, 2007-ben kezd\u0151d\u00f6tt \u00e9s 2022-ben z\u00e1rul\u00f3 vizsg\u00e1lat a New York-i Columbia Egyetemen egy v\u00edrusellenes gy\u00f3gyszert tesztel olyan betegeken, akiknek h<\/s>erpes simplex v\u00edrus 1 \u00e9s az enyhe Alzheimer-k\u00f3r, hogy kider\u00fclj\u00f6n, a gy\u00f3gyszer k\u00e9pes-e lass\u00edtani a betegs\u00e9g el\u0151rehalad\u00e1s\u00e1t. <\/p>\n\n\n\n

Egyel\u0151re csak annyit tehet\u00fcnk, hogy rem\u00e9lj\u00fck a legjobbakat, \u00e9s v\u00e1rjuk az eredm\u00e9nyeket.<\/p>\n\n\n\n

Ezt az \u00faj elm\u00e9letet m\u00e9g mindig nem fogadta el az eg\u00e9sz tudom\u00e1nyos k\u00f6z\u00f6ss\u00e9g. Sok tud\u00f3s azt \u00e1ll\u00edtja, hogy m\u00e9g ha ez az \u00faj elm\u00e9let be is igazol\u00f3dik, a genetikai t\u00e9nyez\u0151k \u00e9s a rossz mut\u00e1ci\u00f3k tov\u00e1bbra is a betegs\u00e9g f\u0151 okoz\u00f3i maradnak. <\/p>\n\n\n\n

Az Alzheimer-k\u00f3r m\u00e9g mindig egy olyan \u00e1llapot, amelynek val\u00f3di okair\u00f3l nagyon kev\u00e9s tudom\u00e1nyos ismeret \u00e1ll rendelkez\u00e9sre; nincs gy\u00f3gym\u00f3d \u00e9s kev\u00e9s ter\u00e1pi\u00e1s kezel\u00e9si lehet\u0151s\u00e9g. Ha ez az elm\u00e9let v\u00e9g\u00fcl igaznak bizonyul, ez lehet az els\u0151 l\u00e9p\u00e9s a jobb kezel\u00e9sek ir\u00e1ny\u00e1ba. Hajr\u00e1 tudom\u00e1ny!<\/p>\n\n\n\n

Hivatkoz\u00e1sok<\/strong><\/p>\n\n\n\n

ABBOTT, A. A fert\u0151z\u00e9sek vetik el az Alzheimer-k\u00f3r egyes eseteit? Term\u00e9szet<\/strong>, v. 587, n. 7832, p. 22-25, 2020. nov. 4.<\/p>\n\n\n\n

CEC\u00cd, K. et al. Biomarkers in Alzheimer disease. Geri\u00e1tria, gerontol\u00f3gia \u00e9s \u00f6reged\u00e9s<\/strong>, 6. \u00e9vfolyam, 3. sz\u00e1m, 273-282. o., 2012.<\/p>\n\n\n\n

MAKIN, S. Az amiloid hipot\u00e9zis t\u00e1rgyal\u00e1sa. Term\u00e9szet<\/strong>, v. 559, n. 7715, p. S4-S7, 2018. j\u00falius 25.<\/p>\n\n\n\n

YOO, K.-Y.; PARK, S.-Y. Terpenoidok mint potenci\u00e1lis Alzheimer-k\u00f3r elleni ter\u00e1pi\u00e1s szerek. Molekul\u00e1k<\/strong>, v. 17, n. 3, p. 3524-3538, m\u00e1rcius. 2012.<\/p>\n\n\n\n

___
Ha tudom\u00e1nyos c\u00e9lokra kell infografik\u00e1t k\u00e9sz\u00edtenie - mint nekem - az Mind the Graph-ben b\u00e1rmilyen infografik\u00e1t l\u00e9trehozhat, amire sz\u00fcks\u00e9ge van \u00e9s amit csak akar. A platform az\u00e9rt van itt, hogy a tud\u00f3sokb\u00f3l tervez\u0151kk\u00e9 v\u00e1ljanak, hogy n\u00f6velj\u00e9k hat\u00e1sukat! Csatlakozzon hozz\u00e1nk a platformon, kattintson ide<\/a>!<\/p>","protected":false},"excerpt":{"rendered":"

Napjainkban a demenciaesetek t\u00f6bb mint fel\u00e9t az Alzheimer-k\u00f3rhoz sorolj\u00e1k - ez egy kr\u00f3nikus neurodegenerat\u00edv betegs\u00e9g, amely fokozatosan befoly\u00e1solja az ember megismer\u0151 k\u00e9pess\u00e9geit \u00e9s mem\u00f3ri\u00e1j\u00e1t. Napjainkig \u00fagy gondolt\u00e1k, hogy genetikai \u00e9s \u00f6reged\u00e9si t\u00e9nyez\u0151k okozz\u00e1k, a betegs\u00e9g gonosztev\u0151inek \u00e9s f\u0151 b\u0171n\u00f6seinek a [...]<\/p>","protected":false},"author":4,"featured_media":12125,"comment_status":"closed","ping_status":"closed","sticky":false,"template":"","format":"standard","meta":[],"categories":[959,28],"tags":[213,554,964,136],"yoast_head":"\nNew Theory Tries to Explain The Cause of Alzheimer\u2019s<\/title>\n<meta name=\"description\" content=\"Read this blog that discusses a new theory that tries to explain the cause of Alzheimer\u2019s disease. Know what scientists and researchers have found.\" \/>\n<meta name=\"robots\" content=\"index, follow, max-snippet:-1, max-image-preview:large, max-video-preview:-1\" \/>\n<link rel=\"canonical\" href=\"https:\/\/mindthegraph.com\/blog\/hu\/cauza-bolii-alzheimer\/\" \/>\n<meta property=\"og:locale\" content=\"hu_HU\" \/>\n<meta property=\"og:type\" content=\"article\" \/>\n<meta property=\"og:title\" content=\"New Theory Tries to Explain The Cause of Alzheimer\u2019s\" \/>\n<meta property=\"og:description\" content=\"Read this blog that discusses a new theory that tries to explain the cause of Alzheimer\u2019s disease. Know what scientists and researchers have found.\" \/>\n<meta property=\"og:url\" content=\"https:\/\/mindthegraph.com\/blog\/hu\/cauza-bolii-alzheimer\/\" \/>\n<meta property=\"og:site_name\" content=\"Mind the Graph Blog\" \/>\n<meta property=\"article:published_time\" content=\"2021-01-11T13:05:53+00:00\" \/>\n<meta property=\"article:modified_time\" content=\"2023-01-05T17:52:03+00:00\" \/>\n<meta property=\"og:image\" content=\"https:\/\/mindthegraph.com\/blog\/wp-content\/uploads\/2021\/01\/new-theory-tries-to-explain-the-cause-of-alzheimers-disease.png\" \/>\n\t<meta property=\"og:image:width\" content=\"1110\" \/>\n\t<meta property=\"og:image:height\" content=\"600\" \/>\n\t<meta property=\"og:image:type\" content=\"image\/png\" \/>\n<meta name=\"author\" content=\"Fabricio Pamplona\" \/>\n<meta name=\"twitter:card\" content=\"summary_large_image\" \/>\n<meta name=\"twitter:label1\" content=\"Written by\" \/>\n\t<meta name=\"twitter:data1\" content=\"Fabricio Pamplona\" \/>\n\t<meta name=\"twitter:label2\" content=\"Est. reading time\" \/>\n\t<meta name=\"twitter:data2\" content=\"5 minutes\" \/>\n<!-- \/ Yoast SEO plugin. -->","yoast_head_json":{"title":"New Theory Tries to Explain The Cause of Alzheimer\u2019s","description":"Read this blog that discusses a new theory that tries to explain the cause of Alzheimer\u2019s disease. 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He has a Ph.D. and solid scientific background in Psychopharmacology and experience as a Guest Researcher at the Max Planck Institute of Psychiatry (Germany) and Researcher in D'Or Institute for Research and Education (IDOR, Brazil). Fabricio holds over 2500 citations in Google Scholar. He has 10 years of experience in small innovative businesses, with relevant experience in product design and innovation management. 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